Ptic disruption evident in the AD1 cohort via increased concentrations of soluble A or in-creased insoluble A plaque burden. However, whether the effects of APOE*4 on enhanced synaptic binding of toxic soluble A species account for the alterations we observed in the synaptic proteome cannot be concluded from the current experiment. Network Topology as an Indicator of Protein Homeostasis-- When the
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