King place in latent J-Lat 9.2 and 15.4 cell lines, in which the ongoing transcription from the host gene would interfere with transcription from the 5'LTR. Consequently, due to the premature termination of *mRNA at the pA site in the 5'LTR, the 3'LTR would no longer be subjected to TI and could thus be activated. To test this hypothesis, we activated viral gene expression with Tat or TNF- and com