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Nding gendered influences on women's reproductive health in Pakistan: moving beyond the autonomy paradigm. Soc. Sci. Med., 68(7), 1349-56. http://dx.doi.org/10.1016/j.socscimed.2009.01.025 National Institute of Population Studies (NIPS) Islamabad Pakistan Macro International Inc. USA (June 2008). Pakistan Demographic and Health Survey 2006-07. Patton, M. Q. (1999). Enhancing the quality and credib
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D if the HSV-2 diagnosis occurred at or after HIV seroconversion, and ulcers were excluded if they occurred at or after HIV seroconversion. We estimated the proportion of participants with 1 ulcer AE classified as Gradeor above (i.e., moderate, severe, or potentially life-threatening), 1 STI examination during which a perianal ulcer was detected, and 1 STI examination during which a groin ulcer
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Ogether with desmocollin-3, syndecan-4 (SDC4), perlecan, fibulin-2 and laminin-332 as substrates, our data support the hypothesis that MMP10 is involved in the control of keratinocyte migration by breakdown of adhesion com-Molecular Cellular Proteomics 14.MMP10 Substrate Degradomics in KeratinocytesMMPDifferentiationMMP9 DMKNDesmocollinMigration Proliferation AdhesionSyndecan IntegrinsCYR61 4Lam
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Exposes the details to do a birth details check. These days, it is quite possible with the technology advances
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Exposed to autoradiography film. High-mannose moieties were preserved only in untreated virus confirming that PNGase F and endo H effectively cleaved high-mannose residues on GPs of HIV-EBOV virion-like particles. (EPS) Figure S4 Thermolysin treatment of HIV-EBOV GP abrogates enhancement of infection by rhMBL in a thermolysin-concentration dependent manner. We preincubated HIV-EBOV GP virion-like
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Nts in clinical practice but technical optimization are needed to improve quantification. Human immunodeficiency virus (HIV) enters its host cells following the interaction between the virus envelope glycoprotein (Gp120), the cell surface CD4 receptor, and a chemokine receptor, which may be CCR5 and/ or CXCR4, acting as a coreceptor1. The HIV tropism is defined by the coreceptor(s) use and is corr
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Nts in clinical practice but technical optimization are needed to improve quantification. Human immunodeficiency virus (HIV) enters its host cells following the interaction between the virus envelope glycoprotein (Gp120), the cell surface CD4 receptor, and a chemokine receptor, which may be CCR5 and/ or CXCR4, acting as a coreceptor1. The HIV tropism is defined by the coreceptor(s) use and is corr
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Particles mixed withFigure 8. Proposed model of MBL-mediated macropinocytosis of EBOV. MBL carbohydrate recognition domains (CRD) bind to highly glycosylated mucin-rich regions of EBOV GP and the MBL-virion complex is presented to the cell surface. Then MBL binds to cognate cellular receptors, such as C1QBP or calreticulin [6] via MBL collagenous stalks. In this manner, MBL concentrates virus at t
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