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Use of the monitors and asked them to return at 1 week, 4 weeks, and 8 weeks to download data from the monitors. Research assistants did not review recorded adherence data with participants. The protocolEthics StatementEach participant provided written informed consent and the study protocol was approved by the Johns Hopkins Medicine Institutional Review Board (IRB), the University of Maryland IRB

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R JS, Kietz S, Wimalasena J, Gustafsson JA: Estrogen receptor beta inhibits 17beta-estradiol-stimulated proliferation of the breast cancer cell line T47D. Proc Natl Acad Sci USA 2004, 101:1566-1571. 2. Liu MM, Albanese C, Anderson CM, Hilty K, Webb P, Uht RM, Price RH Jr, Pestell RG, Kushner PJ: Opposing action of estrogen receptors alpha and beta on cyclin D1 gene expression. J Biol Chem 2002, 27

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Ate proliferation; uPAR expression also confers resistance to caspase-mediated apoptosis and anoikis (104, 105). Numerous studies have documented that strategies to block uPAR expression are associated with decreased angiogenesis and tumor growth and metastasis (85, 103, 106). In contrast, uPAR has been shown to promote angiogenesis in the eye (107). In the UUO model of chronic kidney disease uPAR