Vels of viral particles, we speculate that average levels of host-viral chimeric transcripts in these cells exceed those in the two J-Lat cell lines. These results thus suggest that TI plays a key role in primary CD4+ T cells. In J-Lat 9.2 and 15.4 cells, the host-viral chimeric transcripts terminated at the pA site in the 5'LTR, but transcripts initiating at the host promoter and terminating at t

Effect of Tat on transcription of the provirus. In agreement with the results in Figure 4, expression of Tat alone exerted minimal stimulatory effect on the 5'LTR (Figures 7A-7C, Tat). However, in combination with TNF-, Tat expression activated viral transcription greatly (70-80 of GFP-expressing cells) in all three cell lines (Figures 7A-7C, Tat + TNF-). We further investigated the ability of PM

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Cial mechanisms. Connell's theory of gender and power has been shown previously to explain the gender effects in the spread of HIV/AIDS infection [11,12]. Sa and Larsen applied this theory in their study in Moshi, Tanzania, using gender inequality to explain women's risk of HIV infection [12]. In this study, we adopt Connell's theory of gender and power as a theoretical framework to explore and cl

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