Ct size shown as differences in proportions with 95 confidence intervals (CI). In the primary analysis, missing viral load data were ignored. However, we also conducted a secondary analysis in which missing viral load data were considered to be failures of viral suppression. For continuous outcomes (log10 HIV RNA level and CD4 cell counts), we used linear regression GEE models to compare the aver

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Ignificantly longer lives. While HAART has been increasing the lifespan of those infected with HIV, it has also led to an increased prevalence of HAD [32-38]. As the pathology of HAD, like Alzheimer's Disease (AD), is commonly characterized by an increase in the amount of amyloid-beta (Ab) peptide in the brain [39], evidence suggesting microglia modulate the clearance of potentially neurotoxic Ab

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With heterotrimeric ) complexes in their inactive state (Bourne et al., 1991; Go( Gales et al., 2006), whereas activation of APP signaling should ?induce their dissociation. Conversely, if APP and APPL function as downstream targets for activated Go , we would not expect trimer. As shown in Figure 3J, them to associate with the antibodies against both the N- and C-terminal domains of APP coimmunop

Incubation forsurvival = live cell number (test)/live cell number (control) ?Page 3 of(page number not for citation purposes)Journal of Translational Medicine 2007, 5:http://www.translational-medicine.com/content/5/1/min at 37 with 1 Triton X-100. Cell lysates were harvested and centrifuged at 1,800 rpm for 5 min. The amount of cell-associated HIV-1 was evaluated using p24 antigen capture ELISA.

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