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S, a link between CD40 and microglia has been established. Upregulation of CD40 expression has been detected on microglia of HIV-1-infected brain tissues [28]. CD40L was also shown to potentiate the ability of HIV-1 Tat to activate monocytes and microglia leading to the overproduction of inflammatory proteins such as cytokines and chemokines [122]. Furthermore HAART is unable to modulate blood bra

Ylation and degradation of IB, leading to activation of NF-B [50, 51]. As shown in Figure 2, free NFB is then competent for translocation and binding to sites in the enhancer region of the HIV-1 long terminal repeat (LTR). The binding of NF-B to the LTR is required for highlevel transcription of HIV-1 RNA and upregulation in the expression of latent reservoir virus. NF-B upregulation of latent pro

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