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Ow-up will be needed to determine whether HCV cure through the use of direct-acting antivirals among HIV/ HCV coinfected elite controllers will make the risk for complications among these patients similar to their HIV monoinfected counterparts. Further studies will also be needed to determine the effects of antiretroviral therapy in this group of patients coupled to its risk/benefit ratio. Whereas

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Tes [26,27]. Once infected or activated by HIV- proteins such as gp120 or Tat, microglia begin to excrete endogenous pro-inflammatory cytokines of the M1 subtype [28]. Histopathologically, activated microglia represent a highly accurate correlate to neuronal death and damage in CNS [29]. Severity of dementia in persons with HAD is strongly correlated with the number of activated macrophages and mi

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Coproteins [1]. Binding of gp120 to the receptor, CD4, on the target cell surface induces major conformational changes in the envelope glycoproteins [2]. These changes allow gp120 to bind the viral coreceptor, either CXCR4 or CCR5 [3?]. CD4 binding also induces the formation of a gp41 pre-hairpin intermediate, in which three hydrophobic grooves on the surface of a coiled coil formed by the heptad