Ollowing the intervention phase) there were no significant differences between DAART and SAT in the proportions with HIV RNA ,50 copies/mL (0.47 vs. 0.33; difference, 0.13; 95 CI, 20.08 to 0.34), the proportions with HIV RNA ,400 copies/mL (0.51 vs. 0.44; difference, 0.08; 95 CI, 20.14 to 0.29), average change in HIV RNA from baseline (21.04 vs. 21.33 log10 copies/mL; difference, 0.29; 95 CI, 2

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Vioral complications [66-69]. Microglia, as a major target of HIV-1 infection in the CNS, are typically a viral reservoir [70-72] and are also key in HIV-1 neuroinvasiveness-penetration into the CNS by the virus [72,73]. Most importantly, a discrepancy between the localization of HIV-infected cells and the severity of neurocognitive symptoms has been described [74-76]. Thus, other mechanisms secon

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Er. Cells were incubated for 1 h at 37 . HIV-1 transcytosis was assessed by detecting the presence of p24 antigen (HIV-1 core profile ELISA) in the basolateral chamber of the transwell. The inhibition of transcytosis was expressed as the percentage of p24 antigen recovered in the basolateral chamber in the presence of each plant lectin by comparison to that recovered without the plant lectins. HIV

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