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Vels of viral particles, we speculate that average levels of host-viral chimeric transcripts in these cells exceed those in the two J-Lat cell lines. These results thus suggest that TI plays a key role in primary CD4+ T cells. In J-Lat 9.2 and 15.4 cells, the host-viral chimeric transcripts terminated at the pA site in the 5'LTR, but transcripts initiating at the host promoter and terminating at t

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Other energetically unfavorable helical disruptions, are counteracted by internal hydrogen bonding between surrounding residues. Further bends or kinks are induced through Gly-Gly, Pro-Pro, or Gly-Pro segments, ensuring that the helical structure is disrupted. In general, the calculated tilt angle for each transmembrane helix (H) is 22 12?(Nyholm et al., 2007). In rhodopsin, the strongest helical