Ignificantly longer lives. While HAART has been increasing the lifespan of those infected with HIV, it has also led to an increased prevalence of HAD [32-38]. As the pathology of HAD, like Alzheimer's Disease (AD), is commonly characterized by an increase in the amount of amyloid-beta (Ab) peptide in the brain [39], evidence suggesting microglia modulate the clearance of potentially neurotoxic Ab

N of cRAI in the past three months being reported more frequently in the placebo arm (P = 0.01). Of the 1,383 participants who tested seronegative for HSV-2 at baseline, 36 (2.6 ) did not contribute person-time to incidence analyses because they were retrospectively found to be HIVinfected at baseline, tested seropositive for HSV-2 at the enrollment visit subsequent to screening, or were lost to f

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Kplace was not reached, probably resulting in underestimation of HIV prevalence. This limitation does not apply to this study, as the sample was sufficient to achieve Markov equilibrium and the homophily effect did not affect the estimated HIV prevalence. The estimate was very close to what had been found previously,28 even using the procedure of post-stratification according to workplace. To conc

Issing: 4) B 10 years ] 10 years Mobility (nights outside/month) (missing: 4) 0 1 to 5 6' Pregnant or breastfeedinga Yes No Medical circumcisionb (missing: 15) Yes No TOTALaNumber of HIV-positive individualsWeighted HIV prevalence ( ' 95 CI)4274 2995 1563 1120 1066 979 828 564 441 411 297 1703 4648 626 262 5444 1820 1740 5526 320 6945 5461 1602 202 1444 2830 295 2651839 394 28 67 154 249 266 186